COVID-19 is predominantly a respiratory disease, the clinical outcomes of severe SARS-CoV-2 infections have been seen to affect multiple organ systems. Endothelial dysfunction, micro thrombosis, and hypercoagulation associated with myocarditis-like presentations of impaired cardiac function have been observed in 20% to 30% of hospitalized COVID-19 cases. Furthermore, vascular remodeling and microthrombi formation are associated with increased cardiac serum markers, severe disease, and mortality.

Despite the rising number of COVID-19 cases with clinical outcomes involving cardiac complications, the mechanisms of cardiac pathology and heart injury during severe SARS-CoV-2 infections remain unclear. Current hypotheses include myocardial and vascular wall damage due to cytokine storm, which is the increased release of proinflammatory cytokines such as interleukins (IL) 1 and 6, tumor necrosis factor-alpha (TNF-α), and interferon-gamma (IFN-γ), and the presence of SARS-CoV-2 viral particles in cardiomyocytes and cardiac macrophages.

International Research Awards on Cardiology and Cardiovascular Medicine

Website: https://cardiology-conferences.pencis.com/awards/

#Coaonary artery 

#Hypertension 

#bloodpressure

#cardio

#cardiology

#Cardiovascular

#Atrial fibirllation

#Pacemaker

#Heartfailure

#Myocardial